|
P8.108 Two
cases of tufted folliculitis
Nicole
Orendain Koch
Department of Dermatology, Resident at Jalisco´s
Dermatology Institute Buenos Aires, Providencia Guadalajara, Jalisco, Mexico
Introduction:
Tufted folliculitis is an
inflammatory, purulent and recurrent progressive disease of the scalp,
producing characteristically multiple hair tufts in the area affected. The
destruction of the follicles produces scarring alopecia. Tufted hair
folliculitis is a rare condition, described for the first time by Tagami in
1970 as "numerous multiple hairs." It affects both sexes with a
male to female ratio of 2.7:1, and has been reported in patients between the
ages of 19 and 68 years.
Case 1: 53 year old Mexican male, presenting with
a 10 year old history of pustules and hair loss. On physical examination he
had a plaque with whitish adherent scales measuring 10x10cm in the right
parietal and occipital region, with pseudoalopecia and the presence of multiple
hair tufts, with 2-10 hair shafts emerging from one follicle.
Case 2: 34 year old Mexican male,
presenting with a 3 year old history of a minor head injury, followed by a
persisting crust with hair loss in the same area. On physical examination the
patient had a scaly whitish plaque of 8x9cm, with the presence of 10-20 hair
shafts in each follicular ostium. Both
patients had positive cultures for Staphylococcus aureus, negative
mycological studies, and responded partially to prolonged treatment with
erythromycin, with neither regression nor progression of the illness.
Commentary:
The etiology of tufted folliculitis
is still unknown - the different hypotheses include: infectious causes (due to
Staphylococcus aureus), retention of telogen hairs, and traumatic
causes, amongst others.The treatment of this relapsing condition leading to
progressive scarring alopecia has been notoriously difficult. Systemic
antibiotics produce only a brief response, without permanent eradication.
Corticosteroids suppress the inflammatory response, but produce only slight
improvement and have no lasting effects after treatment stops. Other treatments
include topical and systemic retinoids, zinc sulphate, rifampicin, as well as
surgery, all without great success. The two cases we reviewed fit in the sex
and age group reported in the literature, and both cases had Staphylococcus
aureus as the causal agent. In the second patient, a history of
trauma was followed by alopecia. In both patients there was no progression of
the disease with treatment (erythromycin). Tufted folliculitis should be
considered a different clinical and histopathological entity within the folliculitis
decalvans, and its early recognition and treatment might prevent the
progression of the illness.
|